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.){ref-type=”fig”}) and the proliferation of *M. bovis* BCG and other species of mycobacteria, as well as the production of IL-10 and TNF-α by macrophages infected with the different strains \[[@B65-pathogens-08-00089]\]. In this study, we have previously reported that *M. tuberculosis* MPP8 is associated with IL-10 production and protection against pulmonary TB in humans \[[@B35-pathogens-08-00089]\]. In this regard, we have shown that the MPP8 peptide A9 binds the macrophage IL-10 receptor, exerting its immunoregulatory role.
Finally, the protective role of MPP8 in mycobacterial infections is not limited to the lungs, but extends to the gut mucosa. In a gnotobiotic mouse model, the oral administration of a *M. bovis* BCG mutant lacking *mpb78* exhibited a similar level of oral infection as the *M. bovis* BCG wild-type strain, indicating that MPP8 is not necessary for mycobacterial establishment in the gut \[[@B66-pathogens-08-00089]\]. Moreover, MPP8 was shown to have a protective role in a murine model of enteritis \
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